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the density $p$ per area; $r_{i} $ is the specific intracellular resistivity.  \subsection {Glial contribution into LFP}  The glial contribution into LFP, $\varphi_G(t)$, \varphi_G(t)$, is proportial to the glial membrane currents of potassium ions, thus  can be approximated as follows: \begin{equation} \label{e2}  \varphi_G(t)=k_G \Biggl[ {d[K^+]_e \over dt} \Biggr]_{Glia} \Biggr]_{Glia},  \end{equation}  where $\Biggl[ {d[K^+]_e \over dt} \Biggr]_{Glia}$ is the potassium concentration change due to glia; $k_G$ is a coefficient.  Potassium currents through neuronal membranes are most pronounced during the spiking activity. That is why, approximately the currents are just proportional to the firing rate of principal neurons, $\nu(t)$. The increase of $[K^+]_e$ is then filtered by glial cells. Thus, the equation for the potassium concentration change due to glia should look like the refractory equation  \begin{equation} \label{e3}  \tau {\varphi_G(t) \over dt } = - \varphi_G(t) + c \nu(t),   \end{equation}  where $\tau$ is the characteristic time constant; $c$ is a coefficient.   The time constant cumulatively express the kinetics of neuronal and glial voltage-gated channels, as well as the scales of glial intracellular processes evoked by neuronal activity.  \subsection {Cumulative LFP model}