Antonio Prestes GarcĂ­a edited bibliography/biblio.bib  over 8 years ago

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@book{citeulike:13778825,  author = {Voet, Donald},  citeulike-article-id = {13778825},  citeulike-linkout-0 = {http://www.amazon.ca/exec/obidos/redirect?tag=citeulike09-20\&path=ASIN/B003707NUU},  citeulike-linkout-1 = {http://www.amazon.de/exec/obidos/redirect?tag=citeulike01-21\&path=ASIN/B003707NUU},  citeulike-linkout-2 = {http://www.amazon.fr/exec/obidos/redirect?tag=citeulike06-21\&path=ASIN/B003707NUU},  citeulike-linkout-3 = {http://www.amazon.jp/exec/obidos/ASIN/B003707NUU},  citeulike-linkout-4 = {http://www.amazon.co.uk/exec/obidos/ASIN/B003707NUU/citeulike00-21},  citeulike-linkout-5 = {http://www.amazon.com/exec/obidos/redirect?tag=citeulike07-20\&path=ASIN/B003707NUU},  edition = {Water Damaged, Highlighted, Notation},  howpublished = {Hardcover},  keywords = {biochemistry},  posted-at = {2015-09-30 10:48:49},  priority = {2},  publisher = {Wiley},  title = {{Biochemistry, 2nd Second Edition}}  }  @book{citeulike:13778820,  author = {Stryer, Lubert},  citeulike-article-id = {13778820},  citeulike-linkout-0 = {http://www.amazon.ca/exec/obidos/redirect?tag=citeulike09-20\&path=ASIN/071673687X},  citeulike-linkout-1 = {http://www.amazon.de/exec/obidos/redirect?tag=citeulike01-21\&path=ASIN/071673687X},  citeulike-linkout-2 = {http://www.amazon.fr/exec/obidos/redirect?tag=citeulike06-21\&path=ASIN/071673687X},  citeulike-linkout-3 = {http://www.amazon.jp/exec/obidos/ASIN/071673687X},  citeulike-linkout-4 = {http://www.amazon.co.uk/exec/obidos/ASIN/071673687X/citeulike00-21},  citeulike-linkout-5 = {http://www.amazon.com/exec/obidos/redirect?tag=citeulike07-20\&path=ASIN/071673687X},  citeulike-linkout-6 = {http://www.worldcat.org/isbn/071673687X},  citeulike-linkout-7 = {http://books.google.com/books?vid=ISBN071673687X},  citeulike-linkout-8 = {http://www.amazon.com/gp/search?keywords=071673687X\&index=books\&linkCode=qs},  citeulike-linkout-9 = {http://www.librarything.com/isbn/071673687X},  edition = {4th},  howpublished = {Hardcover},  isbn = {071673687X},  keywords = {biochemistry},  posted-at = {2015-09-30 10:43:24},  priority = {2},  publisher = {W.H. Freeman \& Company},  title = {{Biochemistry}},  url = {http://www.worldcat.org/isbn/071673687X}  }  @article{citeulike:13777623,  abstract = {{The coupling between chromosome replication and cell division includes temporal and spatial elements. In bacteria, these have globally been resolved during the last 40 years, but their full details and action mechanisms are still under intensive study. The physiology of growth and the cell cycle are reviewed in the light of an established dogma that has formed a framework for development of new ideas, as exemplified here, using the Cell Cycle Simulation (CCSim) program. CCSim, described here in detail for the first time, employs four parameters related to time (replication, division and inter-division) and size (cell mass at replication initiation) that together are sufficient to describe bacterial cells under various conditions and states, which can be manipulated environmentally and genetically. Testing the predictions of CCSim by analysis of time-lapse micrographs of Escherichia coli during designed manipulations of the rate of DNA replication identified aspects of both coupling elements. Enhanced frequencies of cell division were observed following an interval of reduced DNA replication rate, consistent with the prediction of a minimum possible distance between successive replisomes (an eclipse). As a corollary, the notion that cell poles are not always inert was confirmed by observed placement of division planes at perpendicular planes in monstrous and cuboidal cells containing multiple, segregating nucleoids.}},  author = {Zaritsky, Arieh and Wang, Ping and Vischer, Norbert O.},  citeulike-article-id = {13777623},  citeulike-linkout-0 = {http://view.ncbi.nlm.nih.gov/pubmed/21565934},  citeulike-linkout-1 = {http://www.hubmed.org/display.cgi?uids=21565934},  issn = {1465-2080},  journal = {Microbiology (Reading, England)},  keywords = {bacterial-biology},  month = jul,  number = {Pt 7},  pages = {1876--1885},  pmid = {21565934},  posted-at = {2015-09-29 10:10:03},  priority = {2},  title = {{Instructive simulation of the bacterial cell division cycle.}},  url = {http://view.ncbi.nlm.nih.gov/pubmed/21565934},  volume = {157},  year = {2011}  }  @article{citeulike:13777224,  abstract = {{Bacterial biofilms, often composed of multiple species and genetically distinct strains, develop under complex influences of cell-cell interactions. Although detailed knowledge about the mechanisms underlying formation of single-species laboratory biofilms has emerged, little is known about the pathways governing development of more complex heterogeneous communities. In this study, we established a laboratory model where biofilm-stimulating effects due to interactions between genetically diverse strains of Escherichia coli were monitored. Synergistic induction of biofilm formation resulting from the cocultivation of 403 undomesticated E. coli strains with a characterized E. coli K-12 strain was detected at a significant frequency. The survey suggests that different mechanisms underlie the observed stimulation, yet synergistic development of biofilm within the subset of E. coli isolates (n = 56) exhibiting the strongest effects was most often linked to conjugative transmission of natural plasmids carried by the E. coli isolates (70\%). Thus, the capacity of an isolate to promote the biofilm through cocultivation was (i) transferable to the K-12 strain, (ii) was linked with the acquisition of conjugation genes present initially in the isolate, and (iii) was inhibited through the presence in the cocultured K-12 strain of a related conjugative plasmid, presumably due to surface exclusion functions. Synergistic effects of cocultivation of pairs of natural isolates were also observed, demonstrating that biofilm promotion in this system is not dependent on the laboratory strain and that the described model system could provide relevant insights on mechanisms of biofilm development in natural E. coli populations.}},  author = {Reisner, Andreas and H\"{o}ller, Brigitte M. and Molin, S{\o}ren and Zechner, Ellen L.},  citeulike-article-id = {13777224},  citeulike-linkout-0 = {http://dx.doi.org/10.1128/jb.188.10.3582-3588.2006},  citeulike-linkout-1 = {http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1482856/},  citeulike-linkout-2 = {http://view.ncbi.nlm.nih.gov/pubmed/16672612},  citeulike-linkout-3 = {http://www.hubmed.org/display.cgi?uids=16672612},  doi = {10.1128/jb.188.10.3582-3588.2006},  issn = {0021-9193},  journal = {Journal of bacteriology},  keywords = {plasmids},  month = may,  number = {10},  pages = {3582--3588},  pmcid = {PMC1482856},  pmid = {16672612},  posted-at = {2015-09-28 20:45:33},  priority = {2},  title = {{Synergistic effects in mixed Escherichia coli biofilms: conjugative plasmid transfer drives biofilm expansion.}},  url = {http://dx.doi.org/10.1128/jb.188.10.3582-3588.2006},  volume = {188},  year = {2006}  }  @incollection{citeulike:13626283,  abstract = {{Bacterial conjugation is a cell-cell communication by which neighbor cells transmit circular DNA strands called plasmids. The transmission of these plasmids has been traditionally modeled using differential equations. Recently agent-based systems with spatial resolution have emerged as a promising tool that we use in this work to assess three different schemes for modeling the bacterial conjugation. The three schemes differ basically in which point of cell cycle the conjugation is most prone to happen. One alternative is to allow a conjugative event occurs as soon a suitable recipient is found, the second alternative is to make conjugation equally like to happen throughout the cell cycle and finally, the third one technique to assume that conjugation is more likely to occur in a specific point late in the cell cycle.}},  author = {Prestes Garc\'{\i}a, Antonio and Rodr\'{\i}guez-Pat\'{o}n, Alfonso},