Results: Statistical Learning for Metabolites detected in newly diagnosed diabetes patients

Background/Motivation

Glucose dependency renders the developing brain particularly vulnerable to the dysglyacemic insults of type 1 diabetes [1]. Whilst much of the research to date has focussed upon the impacts of diabetic dysglycaemia after diagnosis, recently there has been a focus upon the brain impacts of diabetic ketoacidosis (DKA) [2-5]. DKA can occur at any time but affects up to 15-70An understanding of the exact cellular mechanisms of cerebral oedema has remained elusive- due in part to the infrequency of the problem and the idiosyncratic nature of each clinical episode of resuscitation. In vitro and in vivo animal studies have thus been employed to identify the pathophysiology of