GERD disease that affects many people here may appear in some individuals as a genetic disease where the spread between family members, and rarely eating appetite. “Proposed respiratory contributions to the development of GERD include respiratory medications that may alter esophageal sphincter tone and changes in respiratory mechanics, with increased lung hyperinflation compromising the antireflux barrier” . “Although GERD is usually confined to the lower esophagus in some individuals, it may be associated with pulmonary microaspiration of gastric contents” . And we will explain what are the scents that aggravate GERD disease and the mechanism of this happening between the olfactory receptors and the vagus nerve . GERD is causing a lot of trouble to the people and lead to weight loss and feeling angry for long time. A person who sensitive to odors and so we will explain how this relationship smells norepinephrine hormones for digestion. “Acid and pepsin are known to cause esophagitis” . Through the preview it shows that GERD patients are sensitive to odors and that some of them lead to decreased appetite This means a feeling of fullness . Since a lot of research studies conducted on animals , but I this actual preview on patients with GERD and observe physiological changes in them and that means experience the best proof . This research relies on observation and conclusion.
Why dampen the interest of appetite in patients who suffer GERD, when they are exposed to certain smells?
We'll explain how to affected sensors sensual aromatherapy.
Why do not feel hungry who suffer GERD disease ?
What will you do if you get GERD?
GERD patient 's who does not want food . Why? And how to explain it when a person stays for a long time , which did not feel the desire for food ? Is there a self- sufficient production within the cells of their needs ? Thus, means that certain scents play a role in the proportion Ghrelin, therefore, the person does not feel hungry. Sometimes, the patient feels that a meal is palatable , quickly feel satiety, especially that they contain fatty certain odors.“The ob gene product, leptin, is an important circulating signal for the regulation of body weight” (Tartaglia, Louis A., et al.,1995) . “One pathway leads through cells of the Bowman’s glands into the duct of the gland, with fibroblasts of the lamina propria being a potential intermediary . Amino acid odors are detected by olfactory sensory neurons that express only one out of many olfactory receptor genes. Axons of sensory neurons containing the same olfactory receptor converge on specific glomeruli in the olfactory bulb” [5,6,7]. Therefore, leptin plays a role in regulating energy consumption and from the smells that cling Bowman glands in the nose is extracted primary particles are then assembled with mental fabric crystals to produce enough fatty principals body's need for this patient stays for long — without feeling hungry.
The existence of RNA spectral, as Bio- rays (carries genetic traits, entropy, heat, fractals, rapid exponential growth, attractive, etc.). Neuropathologically, the disease affects the caudate nucleus and the cerebral cortex, and also the hypothalamus. “The recently discovered orexigenic hormone of gastric origin, ghrelin and the adipocyte hormone leptin, are two peripherally produced hormones exerting opposite effects on specific populations of hypothalamic neurons that play a key role in regulating energy intake and energy output” ( Popovic, Vera, et al.,2004)[8,9]. Visceral sensory information is transmitted to the brain through the afferent vagus nerve. “The presence of the ghrelin receptor in vagal afferent neurons was assessed by using reverse-transcription polymerase chain reaction and in situ hybridization histochemistry” (Date, Yukari, et al., 2002) [10,11]. Thus, the rhythm between Hermom secretion of ghrelin, leptin plays a role in cell physiology. When a person is exposed to the greasy odors caused by cooking some chicken or the types of odors emitted from the combustion of certain plants or chewing qat tree leaves, especially that with a dark red color which has a silvery thin membrane. “Ghrelin exhibited gastroprokinetic activity with structural resemblance to motilin and potent orexigenic activity through action on the hypothalamic neuropeptide Y ( NPY) and Y1, receptor which was lost after vagotomy” (Asakawa, Akihiro, et al.,2001). All of the above have an impact on the rhythm of the cells that secrete hormones and cells that contain receptors. When the lack of proportion ghrelin (which should be secreted high quantities before eating) leads to increased secretion of leptin and as a person not eating, the leptin plays a counter role for the hormone NPY, also it produces (leptin) from adtipose tissue of high quantities therefore less energy consumption and thus lose weight.
That Upon arrival odor molecules to olfactory receptors in the nose and then to the hypothalamic and the vagus nerve , where ghrelin receptors coupled GHSR1a and leads to increased secretion of ghrelin. The lay and scientific literature typically claims that humans can discriminate 10,000 odors, but this number has never been empirically validated. We determined the resolution of the human sense of smell by testing the capacity of humans to discriminate odor mixtures with varying numbers of shared components” (Bushdid, Caroline, et al., 2014) . Therefore, the chemical scents play an important role in appetite changes through their effect on the hormone receptor of Ghrelin, it is more than an insult that leptin plays a role in regulating energy consumption, and this is also observed in GERD patients. Other study curtains .Odors splash ability to play an important role in reducing the proportion of Ghrelin Thus, the lack of Ghrelin paid to satiety, and this is what happens in GERD patients.
“Chemosensitivity of mouse tension and mucosal receptors is restricted to a small subpopulation, but these show robust responses consistent with expression of receptors to 5-HT, ATP, low pH, and bile constituents” . Therefore, GERD patients nervous conjugated with appetite disturbance , particularly when exposed to certain smells like smoke produced by the burning of Teucrium polium. The role of hearing receptors in the aggravation of GERD and activity of H.pylori In terms of hearing receptors relationship with GERD we see that there are effects accompany where they are receiving very low- frequencies do not hear it but it translates as Reflex Action such as anger, stomach pain and anxiety. Hearing plays an essential role in the physiology of cells , also auditory gens capabilities evolve in sensing low-frequency below 16 Hz, which comes from thermal objects, where we cannot hear and understand those low-frequencies, but translate electrochemical reading into different flavors, that stimulate or inhibit certain neurological and hormonal processes, in the nerve cells such as the heart. The role of H. pylori in these patients is controversial. “Several trials assessed the potential role of H. pylori eradication in improving dyspeptic symptoms, and data of some meta-analyses demonstrated that cure of infection is associated with a small (10%), but significant therapeutic gain as compared to placebo” (Zullo, Angelo, et al., 2014) . Therefore, GERD patients showed synchronized rhythm electrochemical energy and the worsening of the disease, and this demonstrates the role that Hearing receptors play in creating a fertile environment for the bacteria and thus the occurrence of indigestion For this to be the outcome of the medicine does not do any good. Audio signals that turn into electrochemical energy play a role in nerve physiology as well as affected by several factors related to the transition pathways to another phase. This is apparently the GERD patients, particularly the interaction of facilities during inflammation of the urinary system. “The SDNN index, 5-min total power, very low frequency (VLF) and low frequency (LF) power were significantly lower in panic patients relative to controls over the 24-h period. Hourly means were significantly lower during some of the waking hours as well as the latter part of the sleep cycle. In contrast, the mean RR interval, RMSSD and high frequency (HF) power were comparable in patients and controls”. “Results suggest that sympathetic activity is depressed in PD patients under usual life conditions, leading to a relative predominance of vagal tone” (McCraty, Rollin, et al.,2001), [16, 17, 18,19]. Therefore, that the aggravation of the pain associated with these bacteria have simultaneous rhythmic way with electrochemical signals. The infection with these bacteria be the result of exposure to wavelengths electrochemical which in turn raise the entropy of vagus nerve, and thus increases the activity of bacteria. It may be synthesized by finding complementary molecular appropriately in accordance with the self- synthesis by complementary molecular, providing there is a fertile environment, to form a placental synthesis. Therefore, when the evolution of hearing and photoreceptors be together as a coalition of entanglement plays a role in sensory reception of low-frequency waves, and it is the performance of the coalition based on molecules derived from food, drink and ideas as well. When these molecules are negative, it will lead to the emergence of many diseases such as “H. pylori” and schizophrenia; it contains molecules responsible for attracting low-frequency waves.