Results
Fifty percent of the CRSwNP group had deletion of the hld gene which may
promote small colony variant formation or reduced enterotoxin
production, and seventy-five percent expressed virulence genes
associated with invasive disease. The CRSwNP isolate had a superior
ability to localise intracellularly at 6 and 9 hours and showed a higher
burden of S. aureus colony forming units at 24 hours.