INTRODUCTION
Ventricular arrhythmias manifest as a considerable etiological factor contributing to sudden cardiac death, underpinning the necessity to comprehend associated risks¹. Ventricular tachycardia, delineated by the manifestation of wide complex tachycardia, is characterized by a rate exceeding 100 beats per minute with three or more consecutive beats, thereby substantiating its clinical definition². The manifestation of this condition can either be sustained or non-sustained, with the former denoting durations exceeding 30 seconds²
These classifications of arrhythmias are implicated in a substantial proportion of sudden cardiac death incidents in the United States, accounting for the majority¹, ³. Moreover, they represent 8% of all recorded instances of wide complex tachycardias⁴. Structural heart disease is predominantly linked to ventricular tachycardia⁵, and a significant correlation with coronary artery disease is also evident⁶. As such, the imperative nature of discerning the risks correlated with this pathological condition is clear. Anemia has been demonstrated to affect outcomes of atrial fibrillation7,8,9 and studies showed its association with supraventricular tachycardia10. Anemia is also known to increase cardiovascular morbidity and mortality11. Although There is poor data correlating anemia and ventricular tachycardia.
In this study, the authors aim to study the outcomes in terms of mortality, length of stay and total hospital charges in patients with a primary diagnosis of Ventricular Tachycardia and secondary diagnosis of anemia.