INTRODUCTION
Ventricular arrhythmias manifest as a considerable etiological factor
contributing to sudden cardiac death, underpinning the necessity to
comprehend associated risks¹. Ventricular tachycardia, delineated by the
manifestation of wide complex tachycardia, is characterized by a rate
exceeding 100 beats per minute with three or more consecutive beats,
thereby substantiating its clinical definition². The manifestation of
this condition can either be sustained or non-sustained, with the former
denoting durations exceeding 30 seconds²
These classifications of arrhythmias are implicated in a substantial
proportion of sudden cardiac death incidents in the United States,
accounting for the majority¹, ³. Moreover, they represent 8% of all
recorded instances of wide complex tachycardias⁴. Structural heart
disease is predominantly linked to ventricular tachycardia⁵, and a
significant correlation with coronary artery disease is also evident⁶.
As such, the imperative nature of discerning the risks correlated with
this pathological condition is clear. Anemia has been demonstrated to
affect outcomes of atrial fibrillation7,8,9 and
studies showed its association with supraventricular
tachycardia10. Anemia is also known to increase
cardiovascular morbidity and mortality11. Although
There is poor data correlating anemia and ventricular tachycardia.
In this study, the authors aim to study the outcomes in terms of
mortality, length of stay and total hospital charges in patients with a
primary diagnosis of Ventricular Tachycardia and secondary diagnosis of
anemia.