Figure 2: Host and parasite development in live D. magna (left) and ephippia (right) treatments by temperature. For live treatments, broadly resistant clones are in black and broadly susceptible clones are in grey. D. magna development is measured in the time to maturation based on the average day (+/- standard error) in a jar when an individual first developed eggs (a-b). P. ramosa development is shown by the final (at experiment end) prevalence with the number of jars (out of 20) visibly infected for each temperature and resistotype (c-d), and by the average number (+/- standard error) in days until an individual looked infected for each jar (e-f). For both ephippia and live D. magna treatments, time to maturation and time to infection were negatively associated with temperature. Within live treatments, an interaction between resistotype and temperature was detected for prevalence, with the slope of the temperature-prevalence relationship being steeper for broadly resistant clones. An additional main effect of resistotype was detected for time to infection, with susceptible clones becoming infected faster than broadly resistant ones.