At present, the patient was admitted to our hospital due to frequent ICD shocks. His ICD record showed rapid monomorphic VT (mean cycle length, 185 ms), which was always triggered by the same premature ventricular contraction (PVC) (Figure 2). The morphology of the PVC was a left bundle branch block configuration with an inferior axis, and catheter ablation to the trigger PVC was performed. A three-dimensional (3D) voltage map of the right ventricle was constructed, and a low-voltage area (LVA) on the free wall of the right ventricular outflow tract (RVOT) was detected. A good pacemap was obtained at the border zone of the LVA, and the target PVC was ablated at the site. Shortly after discharge, he experienced the recurrence of ICD shocks and underwent a second ablation session the following month. However, the ablation failed again, and he revisited our hospital a month after the second session due to frequent ICD shocks. Monomorphic VT originating from the RVOT was recorded on an electrocardiogram (Figure 3A), and the third ablation session for recurrent VT was performed. The morphology of the VT was the same as the trigger PVC, which was frequently observed during his sessions. An almost perfect pacemap (score, 96) with PVC was obtained near the LVA, slightly posterior to the prior ablation site. The ventricular potential of the PVC on the ablation catheter showed up earlier than the QRS onset of any leads on the electrocardiogram at this site (Figure 3B–C). Both VT and PVC were successfully eliminated and not inducible after ablation. There were no ICD shocks one year after the last session.